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Document Title Author Full Name Author Short Name Index Corresponding Address ResearcherID ResearcherID Author Name ORCID ORCID Author Name Related Email
Isolation and Characterization of Four Unreported Penicillium Species Isolated from the Freshwater Environments in Korea Jung, Hee-Young Jung, HY 8 Kyungpook Natl Univ, Dept Plant Med, Daegu, South Korea leesy1123@knu.ac.kr;
Isolation and Characterization of Four Unreported Penicillium Species Isolated from the Freshwater Environments in Korea Jung, Hee-Young Jung, HY 8 Kyungpook Natl Univ, Inst Plant Med, Daegu, South Korea leesy1123@knu.ac.kr;
Isoquercitrin Suppresses Esophageal Squamous Cell Carcinoma (ESCC) by Inducing Excessive Autophagy and Promoting Apoptosis via the AKT/mTOR Signaling Pathway Liu, Zhibin Liu, ZB 1 Kyungpook Natl Univ, Res Inst Innovat Anim Sci, Dept Anim Sci & Biotechnol, Sangju Si 37224, Gyeongsangbuk D, South Korea AAL-6838-2021 Liu, Zhibin liuzhibin1124@naver.com; hk842767619@gmail.com; huanghai1227@126.com; wjddn5460@naver.com; jenus4@naver.com; yyok1012@naver.com; djkim@hci-cn.org; silee78@knu.ac.kr; sangsik@knu.ac.kr; kdy51311@korea.kr; kdliu@zzu.edu.cn; jaewoong64@knu.ac.kr; mhlee@dsu.ac.kr; leima7597@knu.ac.kr; ok4325@knu.ac.kr;
Isoquercitrin Suppresses Esophageal Squamous Cell Carcinoma (ESCC) by Inducing Excessive Autophagy and Promoting Apoptosis via the AKT/mTOR Signaling Pathway Huang, Ke Huang, K 2 Kyungpook Natl Univ, Res Inst Innovat Anim Sci, Dept Anim Sci & Biotechnol, Sangju Si 37224, Gyeongsangbuk D, South Korea liuzhibin1124@naver.com; hk842767619@gmail.com; huanghai1227@126.com; wjddn5460@naver.com; jenus4@naver.com; yyok1012@naver.com; djkim@hci-cn.org; silee78@knu.ac.kr; sangsik@knu.ac.kr; kdy51311@korea.kr; kdliu@zzu.edu.cn; jaewoong64@knu.ac.kr; mhlee@dsu.ac.kr; leima7597@knu.ac.kr; ok4325@knu.ac.kr;
Isoquercitrin Suppresses Esophageal Squamous Cell Carcinoma (ESCC) by Inducing Excessive Autophagy and Promoting Apoptosis via the AKT/mTOR Signaling Pathway Huang, Hai Huang, H 3 Henan Univ Chinese Med, Tradit Chinese Med Zhong Jing Sch, Henan Int Joint Lab TCM Syndrome & Prescript Signa, Zhengzhou 450046, Peoples R China liuzhibin1124@naver.com; hk842767619@gmail.com; huanghai1227@126.com; wjddn5460@naver.com; jenus4@naver.com; yyok1012@naver.com; djkim@hci-cn.org; silee78@knu.ac.kr; sangsik@knu.ac.kr; kdy51311@korea.kr; kdliu@zzu.edu.cn; jaewoong64@knu.ac.kr; mhlee@dsu.ac.kr; leima7597@knu.ac.kr; ok4325@knu.ac.kr;
Isoquercitrin Suppresses Esophageal Squamous Cell Carcinoma (ESCC) by Inducing Excessive Autophagy and Promoting Apoptosis via the AKT/mTOR Signaling Pathway Kim, Eungyung Kim, E 4 Univ Texas Hlth Sci Ctr San Antonio, Sch Dent, Dept Oral & Maxillofacial Surg, San Antonio, TX 78229 USA liuzhibin1124@naver.com; hk842767619@gmail.com; huanghai1227@126.com; wjddn5460@naver.com; jenus4@naver.com; yyok1012@naver.com; djkim@hci-cn.org; silee78@knu.ac.kr; sangsik@knu.ac.kr; kdy51311@korea.kr; kdliu@zzu.edu.cn; jaewoong64@knu.ac.kr; mhlee@dsu.ac.kr; leima7597@knu.ac.kr; ok4325@knu.ac.kr;
Isoquercitrin Suppresses Esophageal Squamous Cell Carcinoma (ESCC) by Inducing Excessive Autophagy and Promoting Apoptosis via the AKT/mTOR Signaling Pathway Kim, Hyeonjin Kim, H 5 Kyungpook Natl Univ, Res Inst Innovat Anim Sci, Dept Anim Sci & Biotechnol, Sangju Si 37224, Gyeongsangbuk D, South Korea liuzhibin1124@naver.com; hk842767619@gmail.com; huanghai1227@126.com; wjddn5460@naver.com; jenus4@naver.com; yyok1012@naver.com; djkim@hci-cn.org; silee78@knu.ac.kr; sangsik@knu.ac.kr; kdy51311@korea.kr; kdliu@zzu.edu.cn; jaewoong64@knu.ac.kr; mhlee@dsu.ac.kr; leima7597@knu.ac.kr; ok4325@knu.ac.kr;
Isoquercitrin Suppresses Esophageal Squamous Cell Carcinoma (ESCC) by Inducing Excessive Autophagy and Promoting Apoptosis via the AKT/mTOR Signaling Pathway Kim, Chae Yeon Kim, CY 6 Kyungpook Natl Univ, Res Inst Innovat Anim Sci, Dept Anim Sci & Biotechnol, Sangju Si 37224, Gyeongsangbuk D, South Korea liuzhibin1124@naver.com; hk842767619@gmail.com; huanghai1227@126.com; wjddn5460@naver.com; jenus4@naver.com; yyok1012@naver.com; djkim@hci-cn.org; silee78@knu.ac.kr; sangsik@knu.ac.kr; kdy51311@korea.kr; kdliu@zzu.edu.cn; jaewoong64@knu.ac.kr; mhlee@dsu.ac.kr; leima7597@knu.ac.kr; ok4325@knu.ac.kr;
Isoquercitrin Suppresses Esophageal Squamous Cell Carcinoma (ESCC) by Inducing Excessive Autophagy and Promoting Apoptosis via the AKT/mTOR Signaling Pathway Kim, Dong Joon Kim, DJ 7 Dankook Univ, Coll Med, Dept Microbiol, Cheonan 31116, Chungcheongnam, South Korea KDN-8414-2024 Kim, Dong Joon liuzhibin1124@naver.com; hk842767619@gmail.com; huanghai1227@126.com; wjddn5460@naver.com; jenus4@naver.com; yyok1012@naver.com; djkim@hci-cn.org; silee78@knu.ac.kr; sangsik@knu.ac.kr; kdy51311@korea.kr; kdliu@zzu.edu.cn; jaewoong64@knu.ac.kr; mhlee@dsu.ac.kr; leima7597@knu.ac.kr; ok4325@knu.ac.kr;
Isoquercitrin Suppresses Esophageal Squamous Cell Carcinoma (ESCC) by Inducing Excessive Autophagy and Promoting Apoptosis via the AKT/mTOR Signaling Pathway Lee, Sang In Lee, SI 8 Kyungpook Natl Univ, Res Inst Innovat Anim Sci, Dept Anim Sci & Biotechnol, Sangju Si 37224, Gyeongsangbuk D, South Korea liuzhibin1124@naver.com; hk842767619@gmail.com; huanghai1227@126.com; wjddn5460@naver.com; jenus4@naver.com; yyok1012@naver.com; djkim@hci-cn.org; silee78@knu.ac.kr; sangsik@knu.ac.kr; kdy51311@korea.kr; kdliu@zzu.edu.cn; jaewoong64@knu.ac.kr; mhlee@dsu.ac.kr; leima7597@knu.ac.kr; ok4325@knu.ac.kr;
Isoquercitrin Suppresses Esophageal Squamous Cell Carcinoma (ESCC) by Inducing Excessive Autophagy and Promoting Apoptosis via the AKT/mTOR Signaling Pathway Kim, Sangsik Kim, S 9 Kyungpook Natl Univ, Dept Energy Chem Engn, Sangju Si 37224, Gyeongsangbuk D, South Korea liuzhibin1124@naver.com; hk842767619@gmail.com; huanghai1227@126.com; wjddn5460@naver.com; jenus4@naver.com; yyok1012@naver.com; djkim@hci-cn.org; silee78@knu.ac.kr; sangsik@knu.ac.kr; kdy51311@korea.kr; kdliu@zzu.edu.cn; jaewoong64@knu.ac.kr; mhlee@dsu.ac.kr; leima7597@knu.ac.kr; ok4325@knu.ac.kr;
Isoquercitrin Suppresses Esophageal Squamous Cell Carcinoma (ESCC) by Inducing Excessive Autophagy and Promoting Apoptosis via the AKT/mTOR Signaling Pathway Kim, Sangsik Kim, S 9 Kyungpook Natl Univ, Convergence Res Ctr Mech & Chem Engn, 2559 Gyeongsang Daero, Sangju Si 37224, Gyeongsangbuk D, South Korea liuzhibin1124@naver.com; hk842767619@gmail.com; huanghai1227@126.com; wjddn5460@naver.com; jenus4@naver.com; yyok1012@naver.com; djkim@hci-cn.org; silee78@knu.ac.kr; sangsik@knu.ac.kr; kdy51311@korea.kr; kdliu@zzu.edu.cn; jaewoong64@knu.ac.kr; mhlee@dsu.ac.kr; leima7597@knu.ac.kr; ok4325@knu.ac.kr;
Isoquercitrin Suppresses Esophageal Squamous Cell Carcinoma (ESCC) by Inducing Excessive Autophagy and Promoting Apoptosis via the AKT/mTOR Signaling Pathway Kim, Do Yoon Kim, D 10 Gyeongsangbukdo Livestock Res Inst, Yeongju 36052, Gyeongsangbuk D, South Korea liuzhibin1124@naver.com; hk842767619@gmail.com; huanghai1227@126.com; wjddn5460@naver.com; jenus4@naver.com; yyok1012@naver.com; djkim@hci-cn.org; silee78@knu.ac.kr; sangsik@knu.ac.kr; kdy51311@korea.kr; kdliu@zzu.edu.cn; jaewoong64@knu.ac.kr; mhlee@dsu.ac.kr; leima7597@knu.ac.kr; ok4325@knu.ac.kr;
Isoquercitrin Suppresses Esophageal Squamous Cell Carcinoma (ESCC) by Inducing Excessive Autophagy and Promoting Apoptosis via the AKT/mTOR Signaling Pathway Liu, Kangdong Liu, KD 11 China US Henan Hormel Canc Inst, Zhengzhou 450008, Peoples R China 0000-0002-4425-5625 Liu, Kangdong liuzhibin1124@naver.com; hk842767619@gmail.com; huanghai1227@126.com; wjddn5460@naver.com; jenus4@naver.com; yyok1012@naver.com; djkim@hci-cn.org; silee78@knu.ac.kr; sangsik@knu.ac.kr; kdy51311@korea.kr; kdliu@zzu.edu.cn; jaewoong64@knu.ac.kr; mhlee@dsu.ac.kr; leima7597@knu.ac.kr; ok4325@knu.ac.kr;
Isoquercitrin Suppresses Esophageal Squamous Cell Carcinoma (ESCC) by Inducing Excessive Autophagy and Promoting Apoptosis via the AKT/mTOR Signaling Pathway Ryoo, Zae Young Ryoo, ZY 12 Kyungpook Natl Univ, Sch Life Sci, FOUR KNU Creat Biores Grp BK21, Daegu 41566, Gyeongsangbuk D, South Korea AAQ-1573-2020 RYOO, ZAEYOUNG liuzhibin1124@naver.com; hk842767619@gmail.com; huanghai1227@126.com; wjddn5460@naver.com; jenus4@naver.com; yyok1012@naver.com; djkim@hci-cn.org; silee78@knu.ac.kr; sangsik@knu.ac.kr; kdy51311@korea.kr; kdliu@zzu.edu.cn; jaewoong64@knu.ac.kr; mhlee@dsu.ac.kr; leima7597@knu.ac.kr; ok4325@knu.ac.kr;
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