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2024 연구성과별 연구자 정보 (1147 / 2344)

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Document Title Author Full Name Author Short Name Index Corresponding Address ResearcherID ResearcherID Author Name ORCID ORCID Author Name Related Email
IL2-mediated modulation of small extracellular vesicles secretion and PD-L1 expression: a novel perspective for neutralizing immune suppression within cancer cells Lee, Shin Yup Lee, SY 14 Kyungpook Natl Univ, Sch Med, Dept Internal Med, Daegu, South Korea ykm31@dgist.ac.kr;mcbaek@knu.ac.kr;
IL2-mediated modulation of small extracellular vesicles secretion and PD-L1 expression: a novel perspective for neutralizing immune suppression within cancer cells Lee, Shin Yup Lee, SY 14 Kyungpook Natl Univ, Chilgok Hosp, Lung Canc Ctr, Daegu, South Korea ykm31@dgist.ac.kr;mcbaek@knu.ac.kr;
IL2-mediated modulation of small extracellular vesicles secretion and PD-L1 expression: a novel perspective for neutralizing immune suppression within cancer cells Im, Sin-Hyeog Im, SH 15 Pohang Univ Sci & Technol, Dept Life Sci, Pohang, South Korea AAM-9586-2021 Im, Sin-Hyeog ykm31@dgist.ac.kr;mcbaek@knu.ac.kr;
IL2-mediated modulation of small extracellular vesicles secretion and PD-L1 expression: a novel perspective for neutralizing immune suppression within cancer cells Yea, Kyungmoo Yea, K 16 교신저자 DGIST, Dept New Biol, Daegu, South Korea ykm31@dgist.ac.kr;mcbaek@knu.ac.kr;
IL2-mediated modulation of small extracellular vesicles secretion and PD-L1 expression: a novel perspective for neutralizing immune suppression within cancer cells Yea, Kyungmoo Yea, K 16 교신저자 DGIST, New Biol Res Ctr, Daegu, South Korea ykm31@dgist.ac.kr;mcbaek@knu.ac.kr;
IL2-mediated modulation of small extracellular vesicles secretion and PD-L1 expression: a novel perspective for neutralizing immune suppression within cancer cells Baek, Moon-Chang Baek, MC 17 교신저자 Kyungpook Natl Univ, Cell & Matrix Res Inst CMRI, Sch Med, Dept Mol Med, Daegu, South Korea ykm31@dgist.ac.kr;mcbaek@knu.ac.kr;
IL4 receptor targeting enables nab-paclitaxel to enhance reprogramming of M2-type macrophages into M1-like phenotype via ROS-HMGB1-TLR4 axis and inhibition of tumor growth and metastasis Vadevoo, Sri Murugan Poongkavithai Vadevoo, SMP 1 Kyungpook Natl Univ, Sch Med, Dept Biochem & Cell Biol, 680 Gukchaebosang Ro, Daegu 41944, South Korea wjkim@postech.ac.kr;leebh@knu.ac.kr;
IL4 receptor targeting enables nab-paclitaxel to enhance reprogramming of M2-type macrophages into M1-like phenotype via ROS-HMGB1-TLR4 axis and inhibition of tumor growth and metastasis Vadevoo, Sri Murugan Poongkavithai Vadevoo, SMP 1 Kyungpook Natl Univ, Sch Med, Dept Biomed Sci, 680 Gukchaebosang Ro, Daegu 41944, South Korea wjkim@postech.ac.kr;leebh@knu.ac.kr;
IL4 receptor targeting enables nab-paclitaxel to enhance reprogramming of M2-type macrophages into M1-like phenotype via ROS-HMGB1-TLR4 axis and inhibition of tumor growth and metastasis Vadevoo, Sri Murugan Poongkavithai Vadevoo, SMP 1 Kyungpook Natl Univ, Sch Med, CMRI, 680 Gukchaebosang Ro, Daegu 41944, South Korea wjkim@postech.ac.kr;leebh@knu.ac.kr;
IL4 receptor targeting enables nab-paclitaxel to enhance reprogramming of M2-type macrophages into M1-like phenotype via ROS-HMGB1-TLR4 axis and inhibition of tumor growth and metastasis Kang, Yeoul Kang, YL 2 Pohang Univ Sci & Technol POSTECH, POSTECH CATHOLIC Biomed Engn Inst, Dept Chem, 77 Cheongam Ro, Pohang 37673, Gyeongbuk, South Korea 0000-0002-1655-9023 Kang, Yeoul wjkim@postech.ac.kr;leebh@knu.ac.kr;
IL4 receptor targeting enables nab-paclitaxel to enhance reprogramming of M2-type macrophages into M1-like phenotype via ROS-HMGB1-TLR4 axis and inhibition of tumor growth and metastasis Gunassekaran, Gowri Rangaswamy Gunassekaran, GR 3 Kyungpook Natl Univ, Sch Med, Dept Biochem & Cell Biol, 680 Gukchaebosang Ro, Daegu 41944, South Korea wjkim@postech.ac.kr;leebh@knu.ac.kr;
IL4 receptor targeting enables nab-paclitaxel to enhance reprogramming of M2-type macrophages into M1-like phenotype via ROS-HMGB1-TLR4 axis and inhibition of tumor growth and metastasis Gunassekaran, Gowri Rangaswamy Gunassekaran, GR 3 Kyungpook Natl Univ, Sch Med, Dept Biomed Sci, 680 Gukchaebosang Ro, Daegu 41944, South Korea wjkim@postech.ac.kr;leebh@knu.ac.kr;
IL4 receptor targeting enables nab-paclitaxel to enhance reprogramming of M2-type macrophages into M1-like phenotype via ROS-HMGB1-TLR4 axis and inhibition of tumor growth and metastasis Gunassekaran, Gowri Rangaswamy Gunassekaran, GR 3 Kyungpook Natl Univ, Sch Med, CMRI, 680 Gukchaebosang Ro, Daegu 41944, South Korea wjkim@postech.ac.kr;leebh@knu.ac.kr;
IL4 receptor targeting enables nab-paclitaxel to enhance reprogramming of M2-type macrophages into M1-like phenotype via ROS-HMGB1-TLR4 axis and inhibition of tumor growth and metastasis Lee, Seok-Min Lee, SM 4 Kyungpook Natl Univ, Sch Med, Dept Biochem & Cell Biol, 680 Gukchaebosang Ro, Daegu 41944, South Korea wjkim@postech.ac.kr;leebh@knu.ac.kr;
IL4 receptor targeting enables nab-paclitaxel to enhance reprogramming of M2-type macrophages into M1-like phenotype via ROS-HMGB1-TLR4 axis and inhibition of tumor growth and metastasis Lee, Seok-Min Lee, SM 4 Kyungpook Natl Univ, Sch Med, Dept Biomed Sci, 680 Gukchaebosang Ro, Daegu 41944, South Korea wjkim@postech.ac.kr;leebh@knu.ac.kr;
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